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Yes, academic essays and texts are notoriously difficult. Not to mention boring and long! But critical reading can help make texts easier to understand, as well as easier to get through. Here are five strategies designed to help you read critically.

1. Focus on the big picture. Determine what the author thinks, and why. What is the author's argument? Write down the author's purpose (Why write this?), tone (Why write this way?), and thesis (What's her point?) as you read. This may seem time-consuming, but because you won't have to reread the essay you'll actually save time. You can always find details later. Just be sure you know where to find them.

2. Map the passage. In addition to noting the author's purpose, tone, and main idea(s), you should summarize each section of the essay as you read. Force yourself to be concise and precise; limit yourself to one or two short sentences. Again, this will save you time in the long run, especially if you have to return to the reading for a test or final paper. Note the location and relevance of important details, but don't get bogged down. Just try to keep the information straight. Since you know the author's argument, find the author's evidence and assess it. Jot down quick notes in the margins. You're going to forget where the details and evidence are, and your passage map will help you find them much more quickly.

3. Keep a critical distance. Can you tell the difference between the author's opinions and the opinions of people the author is quoting or referencing? And, perhaps more importantly, can you differentiate between those opinions and your own? It's okay to take arguments personally; you're a human being, with beliefs and emotions. But beliefs and emotions are not substitutes for logic. Respond to arguments as logically as possible, using evidence to justify your response. Otherwise, how will we understand why you believe and feel what you do? Confront the essay on its own terms, and address the author's specific assertions. In other words, act as though you are responding to a real person, in real time.

4. Anticipate and predict. Good essays are designed to be predictable. Academic authors write within a tradition and discipline of scholarship, using definitions and concepts that others have defined. Thus, the more critically you read one essay, the easier it will become to read and understand other essays. This, in turn, will save you even more time. If the author goes someplace unexpected in the essay, and you've been reading carefully, it often means one of two things has happened: - You missed something the author's thesis or purpose, the logic of the argument, an allusion, an implication, a hint, foreshadowing, etc. - The author missed something she or he improperly defined the relevant terms, implied the wrong thing, misapplied his or her own evidence, wrote a poorly organized essay, used tired and inappropriate rhetoric, tried to sell books by saying something fashionable, etc. Either way, it's probably important.

5. Read actively. Television, cinema, novels, and even the evening news have all trained us to take information in passively. You are not a sponge. Active reading means asking yourself questions as you read, such as: - What's the author's thesis? Why is the author writing this? (Strategy #1) - What and where is the evidence? (Strategy #2) - How can I justify my response to this? (Strategy #3) - Where is the author going with this? How does this relate to other arguments I've read, in other classes? (Strategy #4)

Remember: academic writing is BORING, LONG, and DIFFICULT. Reading actively will make the writing seem less boring, shorter, and easier, because it keeps you active and engaged. For more critical reading strategies, visit these websites:

This definition of active reading is taken directly from the 2007 Kaplan, Inc., SAT Lesson Book.

It should be noted that some of the content of this document is either adapted from Kaplan, Inc., strategies and methods, or informed by my experiences and personal development as a Kaplan instructor. As I have been a Kaplan instructor for several years, it is difficult for me to differentiate between training and experiences I have had at Kaplan and training and experiences I may have acquired elsewhere. In any event, I owe much thanks to Kaplan, Inc., and the personnel at the Buffalo Kaplan Center.

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There are many different types (classes) of anti-hypertensive medications (medications that lower blood pressure); these include beta-blockers, calcium channel blockers, diuretics (medications that increase urine volume), angiotensin converting enzyme inhibitors (ACE inhibitors), and angiotensin receptor blockers (ARBs). Anti-hypertensives may be used alone or in combination(s) to control blood pressure. Different classes of anti-hypertensives have different effects on erectile function. Inderal (a beta blocker) and hydrochlorothiazide (a diuretic) are known to cause erectile dysfunction, while calcium channel blockers and ACE inhibitors do not seem to affect erectile function. On the other hand, angiotensin receptor blockers (ARBs) such as losartan (Cozaar) and valsartan (Diovan), may actually increase sexual appetite, improve sexual performance, and decrease erectile dysfunction. Therefore, choosing an optimal anti-hypertensive combination is an important part of treating erectile dysfunction Fig. S1 shows that PDE-5 inhibitors reduce tumor growth but that persistent tumors show evidence of immune editing. In Fig. S2, the antitumor effect of sildenafil is maintained even with delayed treatment. Fig. S3 shows that sildenafil increases CD8+ TILs. Fig. S4 depicts sildenafil as it fails to induce an antitumor effect in 4T1-HA–bearing Rag-2–/– mice. In Fig. S5, sildenafil fails to augment proliferation of purified T cells. Fig. S6 shows that sildenafil does not alter IL-13 or IFN- production from CD11b+ cells. Online supplemental material is available at http://www.jem.org/cgi/content/full/jem.20061104/DC1. Experts believe that psychological factors such as stress, anxiety, guilt, depression, low self-esteem, and fear of sexual failure cause 10 to 20 percent of ED cases. Men with a physical cause for ED frequently experience the same sort of psychological reactions (stress, anxiety, guilt, depression). Other possible causes are smoking, which affects blood flow in veins and arteries, and hormonal abnormalities, such as not enough testosterone. The authors declare no conflicting financial interests. Mary Ann Forciea, MD; online canadian viagra When administered in vitro, PDE5 inhibition induces apoptosis in colon carcinoma (15) and chronic lymphocytic leukemia cells (16). To determine whether similar effects could be observed in vivo, we used various transplantable mouse tumors, including CT26WT (a colon carcinoma; Fig. 1 A), the more aggressive variant C26GM (Fig. 1 B), TS/A (a mammary adenocarcinoma; Fig. 1 C), and the MCA203 fibrosarcoma (Fig. 1 D). PDE5 inhibitors were administered starting on the day of tumor challenge. Sildenafil and tadalafil significantly delayed tumor outgrowth by 50 to 70% in immune-competent mice, although all mice ultimately died (Fig. S1, available at http://www.jem.org/cgi/content/full/jem.20061104/DC1). Similar results were obtained even if sildenafil treatment was started on day 7 after tumor challenge in the CT26WT model (Fig. S2). The fact that no difference in tumor outgrowth was seen between early versus late administration of sildenafil suggests that PDE5 inhibition does not appreciably affect the early phases of tumor uptake but rather influences the later stages of tumor outgrowth. Because the addition of sildenafil to cultured CT26WT cells did not increase their apoptosis or affect their doubling time (unpublished data), we conclude that sildenafil does not have a direct antitumor effect but rather interferes in host–tumor interactions. Smoking is the leading cause of COPD, responsible for between 80 percent and 90 percent of all cases in the United States. 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